Characterization of the Biochemical Pathway of Apoptosis Induced by Three D­glucopyranoside Derivatives from Tulbaghia Violacea

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Date
2014-12
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Abstract
Aim: Chinese hamster ovary (CHO) cells were used to identify the most likely apoptotic biochemical pathway induced by Methyl-α-D-glucopyranoside (MDG), D-fructofuranose- β(2→6)-methyl-α-D-glucopyranoside (DFMDG) and β-D-fructofuranosyl-(2→6)-α-D- glucopyranoside (DFDG) treatments isolated from wild garlic (Tulbaghia violacea). Methodology: Assays, which detect different morphological and biochemical changes in the CHO cells were used to examine the apoptosis induction by individual agents, paying special attention to the mitochondria membrane potential (∆ψm) depolarization. Results: The major events of apoptosis, namely, translocation of phosphatidylserine, mitochondria membrane potential (∆ψm) depolarization and activation of caspase-3 in CHO cells were initiated within 2 hours of treatments. The onset of the apoptosis induction was indistinguishable among the MDG, DFMDG and DFDG, and similar to that of staurosporine. Conclusion: The study shows that CHO cells treated with MDG, DFMDG and DFDG undergo apoptotic death through the mitochondrial (intrinsic) pathway.
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Keywords
Apoptosis, Mitochondrial pathway, Glucopyranosides, Mitochondrial permeability transition pore
Citation
Lyantagaye, S.L., 2014. Characterization of the Biochemical Pathway of Apoptosis Induced by D-glucopyranoside Derivatives from Tulbaghia violacea. Annual Research & Review in Biology, 4(6), p.962.